I think the current hypothesis is that ARBs could enhance COVID-19 reactions:
"The connection between ARBs and COVID-19
Professor Murray Esler is the senior director of the Baker Heart and Diabetes Institute and consultant cardiologist at the Alfred Hospital Melbourne and Adjunct Professor of Medicine at Monash University.
Professor Esler told newsGP he first became concerned about ARBs and COVID-19 when talking to his daughter, a public health physician in the Northern Territory, a few weeks ago.
‘The first clue was really that severe COVID-19 infection was more common in hypertension, and hypertensive patients are really not predisposed to infection at all, so that was very odd,’ he said.
‘So that suggested perhaps this predisposition to severe COVID-19 infection in hypertension could possibly be a drug effect mediated by ARBs which upregulates ACE-2, and potentially predisposes to infectivity but also in particular, severe infection.’
Professor Esler has been in contact with colleagues in Lombardy, Italy, who are currently exploring it further."